The interaction of mosquito immune system with Plasmodium is critical in determining the vector competence.
Human host feeding was confirmed in Anopheles stephensi (30 %), Anopheles subpictus (27 %), Anopheles jamesii (22 %), Anopheles annularis (26 %), and Anopheles nigerrimus (16 %).
Potential targets of Plasmodium ookinetes at the mosquito midgut walls were investigated in relation to interfering malarial transmission.
Genetic engineering technologies can be used both to create transgenic mosquitoes carrying antipathogen effector genes targeting human malaria parasites and to generate gene-drive systems capable of introgressing the genes throughout wild vector populations.
These analyses confirmed morphological features previously described using electron microscopy and uncovered a high degree of individual variation in SG structure. Our studies provide evidence for two alternative models for the origin of the salivary duct, the structure facilitating parasite transport out of SGs.
Malaria is a life-threatening disease caused by parasites transmitted to people and animals through the bites of infected mosquitoes.
The insulin-like peptides (ILPs) and their respective signaling and regulatory pathways are highly conserved across phyla.
Plant-borne compounds can be employed to synthesize mosquitocidal nanoparticles that are effective at low doses.
With the growth of resistance to overused insecticides, vector management has become highly problematic.
Anopheles (Cellia) stephensi Liston 1901 is known as an Asian malaria vector.